At a glance
Alopecia means pathological hair loss. The most common forms are:
Androgenetic alopecia (AGA) — hereditary hair miniaturization in men and women.
Alopecia areata (AA) — sudden round/oval patches of hair loss caused by an autoimmune process.
Early evaluation by a dermatologist–trichologist is key to distinguish these from telogen effluvium, fungal infection, traction, or scarring alopecias.
Men: bitemporal recession, crown thinning (Norwood–Hamilton scale).
Women: diffuse thinning over the crown with a preserved frontal hairline (Ludwig pattern).
Progressive miniaturization: hairs become thinner/shorter; scalp usually non-inflamed.
One or more smooth, sharply bordered patches without scarring.
“Exclamation mark” hairs at the border; may involve eyebrows/eyelashes or beard.
Relapsing–remitting course; can progress to total scalp loss (totalis) in some cases.
Red flags: pain/itch with redness or crusts, scarring, rapid complete shedding, broken hairs with scaling in children (possible tinea capitis). Seek prompt care.
AGA: genetic predisposition and follicle sensitivity to dihydrotestosterone (DHT).
AA: autoimmune attack on hair follicles; often associated with stress, infections, or other autoimmune diseases.
Contributors to evaluate: iron deficiency (low ferritin), thyroid disease, protein deficiency, medications, pregnancy/menopause shifts, tight hairstyles/chemical overprocessing.
Specialist scalp exam and trichoscopy (dermatoscopy of scalp).
Labs as indicated: CBC, ferritin/iron, TSH (thyroid), vitamin D/B12; selective hormonal workup in women.
Rule out tinea capitis and scarring dermatoses.
Baseline photos/trichogram for monitoring.
Results require consistency; meaningful changes are usually seen after 3–6 months and need maintenance.
Topical minoxidil (5% foam/solution) — first line for men and women.
Oral finasteride/dutasteride for men (prescription; discuss adverse effects and contraception for partners of women who may become pregnant).
Antiandrogens for women (e.g., spironolactone) when appropriate and monitored by a specialist.
Adjuncts: PRP (platelet-rich plasma), microneedling, low-level laser therapy.
Hair transplantation (FUE/FUT) once disease is stable and donor supply is adequate.
Supportive care: gentle shampoos, UV scalp protection, correction of nutrient deficiencies.
Intralesional corticosteroid injections (first-line for limited patches).
Topical corticosteroids or contact immunotherapy (e.g., DPCP) per protocol.
JAK inhibitors for moderate–severe disease under specialist supervision.
Topical minoxidil as an adjunct for regrowth.
Psychosocial support; cosmetic camouflage (fibers, micro-pigmentation) when desired.
AGA: aim to slow progression and partially thicken hair; treatment is long-term/ongoing.
AA: spontaneous regrowth is possible, but relapses are common; plan regular follow-ups.
Ensure adequate protein and iron intake; correct deficiencies.
Avoid tight hairstyles, harsh chemical treatments, and excessive heat styling.
Manage stress; prioritize sleep.
Protect the scalp from sun (hats, UV sprays).
Sudden clumps of shedding, children with scaly patches/broken hairs, signs of infection/scarring, intense itch/pain, rapid loss of brows/lashes.
Systemic clues: fatigue, pallor, cold extremities (possible iron deficiency), weight or heart-rate changes (thyroid).
Start with a dermatologist–trichologist. As needed: endocrinologist, gynecologist/andrologist, psychologist. Children: pediatrician + dermatologist.
Typical pathway: consultation, trichoscopy, targeted labs; as indicated — PRP/microneedling, intralesional therapy, laser, or transplant planning. On Doctor911.am you can compare clinics, packages, and current offers, and book a visit.
Do vitamins stop hair loss?
Only if you have a proven deficiency. Routine “hair vitamins” without deficits rarely help.
Do I need minoxidil forever?
For AGA, yes — stopping usually returns hair to its genetic trajectory over months.
Can home remedies replace treatment?
Scalp massage or mild tonics may improve comfort/circulation but do not replace evidence-based therapy.
Is there a single “AGA test”?
No. Diagnosis is clinical with trichoscopy and by excluding other causes.
Will a transplant fix it permanently?
Transplant redistributes hair; AGA continues in non-transplanted areas, so maintenance therapy remains important.
This article is informational and does not replace medical advice. If you notice rapid hair loss, scaly patches in a child, signs of infection, or scarring, arrange an in-person evaluation.